Selective thermal ablation of cancer cells with ferromagnetic nanocontainers

Stały URI zbioruhttp://hdl.handle.net/11652/5397

CONTENTS
1. Selective thermal ablation of cancer cells by ferromagnetic containers Kołaciński Z., Frączyk J., Majsterek I., Kamiński Z., Mościcki A., Przybyłowska-Sygut K., Raniszewski G., Szymański Ł., Pietrzak Ł., Wiak S. ........................................................................................ 7
2. Synthesis of carbon ferromagnetic filled nanocontainers using CVD technique Pietrzak Ł., Wąs Ł., Wyczechowski M. ........................................................................................ 15
3. Natural ceramic nanoparticles ‒ layered aluminosilicates as an active phase of polymer nanocomposites and their potential application in medicine Stodolak-Zych E., Dzierzkowska E., Rapacz-Kmita A. ........................................................................................ 23
4. Mathematical Model of Power Losses with Regard to Magnetic Fluid Hyperthermia Treatment Miaskowski A. ........................................................................................ 32
5. New strategies targeting the unfolded protein response (PERK signaling) for cancer treatment Pytel D., Szulc Z. M., Mucha B., Majsterek I., Diehl J. A. ........................................................................................ 41
6. Functionalization of carbon nanomaterials Mitura K.A., Szczypiński M. M., Korn Ł., Czerwińska E., Kolesińska B. ........................................................................................ 47

Przeglądaj

Filtry

20181

Ustawienia

Autorsearch.filters.applied.operator.equals: search.filters.author.Diehl, J. A.

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  • Pozycja
    New strategies targeting the unfolded protein response (PERK signaling) for cancer treatment
    (Wydawnictwo Politechniki Łodzkiej, 2018) Pytel, D.; Szulc, Z. M.; Mucha, B.; Majsterek, I.; Diehl, J. A.
    "Cancer cells exhibit increased metabolism and proliferation rates. The UPR (Unfolded Protein Response) has garnered attention as a signaling network utilized by cancer cells that contributes to a number of oncogenic processes. The UPR activates three signal transducers (ATF6, IRE1 and PERK); of these the kinase PERK can both potentiate cell survival during moderate stress and provide a mechanism for recovery if cells do not commit to an apoptotic fate. Thus, dissection of the molecular mechanisms whereby PERK signaling influences."